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Indonesian Journal of Cardiology

ijc
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Published by Indonesian Heart Association

ISSN : 28303105 EISSN : 29647304 DOI : -

Core Subject : Health,

Health Professions Medicine & Pharmacology

Indonesian Journal of Cardiology (IJC) is a peer-reviewed and open-access journal established by Indonesian Heart Association (IHA)/Perhimpunan Dokter Spesialis Kardiovaskular Indonesia (PERKI) [www.inaheart.org] on the year 1979. This journal is published to meet the needs of physicians and other health professionals for scientific articles in the cardiovascular field. All articles (research, case report, review article, and others) should be original and has never been published in any magazine/journal. Prior to publication, every manuscript will be subjected to double-blind review by peer-reviewers. We consider articles on all aspects of the cardiovascular system including clinical, translational, epidemiological, and basic studies. Subjects suitable for publication include but are not limited to the following fields: Acute Cardiovascular Care Arrhythmia / Cardiac Electrophysiology Cardiovascular Imaging Cardiovascular Pharmacotherapy Cardiovascular Public Health Policy Cardiovascular Rehabilitation Cardiovascular Research General Cardiology Heart Failure Hypertension Interventional Cardiology Pediatric Cardiology Preventive Cardiology Vascular Medicine

Arjuna Subject : Kedokteran - Cardiology and Cardiovascular Medicine

Articles 608 Documents

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What Is the Cause of Non-responders in CRT and How to Identify It? Yamin, Muhammad
Indonesian Journal of Cardiology Vol. 38, No. 2 April-June 2017
Publisher : The Indonesian Heart Association

No Abstract

Hypolipidemic Effect and Antioxidant Activity of Tamarind Leaves Extract in Hypercholesterol-Fed Rats Aprilia, Citra Ayu; Ninditasari, Ghina; BR, Djoko Walujo
Indonesian Journal of Cardiology Vol. 38, No. 2 April-June 2017
Publisher : The Indonesian Heart Association

Background: Higher cost and side effects made of some anticholesterol drugs usedin long time are the reasons why some people change to herbal therapies. Tamarind(Tamarindus indica) leaves is one of the herbal therapies. This research aims to determinehypolipidemic effect and antioxidant activity of extract of tamarind leaves (ETL).Methods: We used 25 rats as samples, divided into five groups of negative control(CMC 0.5%), positive control (Ezetimibe 1.26 g/kgBW)), first, second and third doseof ETL consequently are 0.93, 1.86 and 3.73 g/kgBW.Results: Paired-samples T-test showed ETL significantly decreased total cholesterol(TC), triglyceride (TG) level, and high-density lipoprotein cholesterol (HDL-C) levelsignificantly increased compared with negative control groups (p≤0.05). Low-densitylipoprotein cholesterol (LDL-C) level had significant difference only at second dose ofETL (p<0.05). Furthermore, the data’s difference between pre- and post-interventionwere analyzed with one-way ANOVA test in TC, TG, and HDL-C level, ETL had asignificant difference (p≤0.05), while there was no significant difference in LDL -Cbetween groups (p>0.05). Data were also analyzed by Post Hoc test. TC, TG, andHDL-C level had a significant difference between all variance ETL’s doses and positivecontrol compared with negative control group (p≤0.05). For antioxidant activity, ETLexhibited the significant reduction in the levels of malondialdehyde (MDA) by pairedsamplesT-test (p≤0.05) but there was no significant difference in both of MDA andsuperoxide dismutase (SOD) level (p>0.05) analyzed by One-way ANO VA test.Conclusion: All variant of ETL’s doses have hypolipidemic effect and antioxidant activity.ETL also has similar effect with Ezetimibe. Saponin, flavonoid, epicatechin, tanin, andpolyphenol that is contained likely contribute to these pharmacologic effects.

Shock Index as Simple Clinical Independent Predictor of In-hospital MACEs in NSTEMI Patients Presenting with Heart Failure Handayani, Ahmad; Kaban, Kartika; Nasri, Marwan; Mukhtar, Zulfikri; Siregar, Abdullah Afif
Indonesian Journal of Cardiology Vol. 38, No. 2 April-June 2017
Publisher : The Indonesian Heart Association

Background: Identification of Non-ST Elevation Myocardial Infarction (NSTEMI) patientsat higher risk of in-hospital complications is very important. Such identification will givecrucial information in determining treatment strategy especially for those come with heartfailure. One of the simple predictor for short term prognosis in acute coronary syndromeis shock index (SI), which is the ratio of heart rate over systolic blood pressure on admission.There had not been any study conducted to evaluate the use of SI in NSTEMI patientscome with heart failure. The aim of this study is to evaluate the SI compared with otherroutine clinical and laboratory examination as a predictor of in-hospital major adversecardiac events (MACEs) in NSTEMI patients presenting with heart failure.Methods: We performed a retrospective analysis of NSTEMI patients with heart failureadmitted to Haji Adam Malik General Hospital in Medan from January 2014 until July 2015.SI was calculated as the ratio of heart rate over systolic blood pressure on presentation.Patients presenting with cardiogenic shock were excluded.Results: There were 55 patients eligible in this study. In-hospital MACEs was found in 24patients (44%) compared with 31 patients (56%) without in-hospital MACEs. Patientswith in-hospital MACEs were older (60.6±10.8 vs. 57.2±7.9, p=0.178), had less historyof dyslipidemia [8(33%) vs. 19 (61%), p=0.032], faster heart rate (111.4±35.8 vs.96.5±24.3, p=0.032], higher GRACE score [139(98-187) vs. 120 (91-148); p=0.001],and higher SI [0.83(0.57-1.5) vs. 0.67 (0.38-1.27), p=0.013). SI >0.8 was the only independentpredictor of MACEs in NSTEMI patients presenting with heart failure (OR=4.3,CI=1.247-14.328, p=0.048).Conclusion: Beyond other routine examinations, SI is the only independent predictor ofin-hospital MACEs in NSTEMI patients presenting with heart failure.

Pemberian Agen Vasoaktif Berdasarkan Hemodinamik pada Syok Kardiogenik Alsagaff, Muhammad Yusuf; Djihan, Laily
Indonesian Journal of Cardiology Vol. 38, No. 2 April-June 2017
Publisher : The Indonesian Heart Association

Cardiogenic shock is a life-threatening emergency that occurs frequently with acutemyocardial infarction (AMI) and the mortality remains over 50% in most studies. Despitesuccessful revascularization, cardiovascular failure leading to multiple organ failure mayoccur. Therapy with vasoactive agents should be initiated to restore adequate arterialpressure and organ perfusion in patients with shock. Recent analysis suggests that systemicinflammatory response syndrome (SIRS) is an important component of the hemodynamicinstability in cardiogenic shock. Inflammation through the nitric oxide (NO) pathway leadingto decrease in vascular resistance and these patients may necessitate supplementalvasopressor therapy. A subanalysis of a prospective randomized trial suggested that norepinephrine(NE) may be preferred over dopamine in patients with cardiogenic shock,while dobutamine is the inotrope of choice. We present a case of a shock cardiogenicand a review of a therapeutic scheme for the pharmacological treatment of patients incardiogenic shock.

Comparison of Roles between Alcohol Ablation and Surgical Myectomy in Hypertrophic Cardiomyopathy Kusuma, Nico; Pranata, Raymond; Vania, Rachel; Siswanto, Bambang Budi
Indonesian Journal of Cardiology Vol. 38, No. 2 April-June 2017
Publisher : The Indonesian Heart Association

Hypertrophic cardiomyopathy is a genetic myocardial disease that may extend to leftventricular outflow tract obstruction. Alcohol septal ablation (ASA) is preferred for itsnon-surgical, agreed as the safest way for advanced aged, or those with high risk for surgicalprocedure or comorbidities. On the other hand, surgical myectomy (SM) remainsas gold standard, established to be the most consistent to achieve optimal hemodynamicand spare longevity, regardless selective use. This article reviews the advantages anddisadvantages between SM and ASA ablation from its outcome, mortality, pre- and postprocedural,and patient’s profile selection.

Calcium Serum Levels and Blood Pressure Response in trained subjects who consumed goat milk Yusni, Yusni; Akbar, Ieva Baniasih; Akbar, M. Rizki
Indonesian Journal of Cardiology Vol 38 No 3 (2017): July - September 2017
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (0.036 KB)

Background: Calcium plays a role in regulating blood pressure and one exogenous sources of calcium are goat milk. Indonesian society is generally believed that goat milk can lower blood pressure and useful as antihypertensive, but so far have not found scientific evidence of how the mechanism of goat milk for controlling blood pressure. This study aimed to analyze the effect of the consumption of goat milk for lowering blood pressure and its relation to calcium serum levels in people trained. Method: Subjects, 19 gymnasts (the treatment group) and 10 runners (the control group), male and female, aged 17-28 years. Treatment: goat’s milk 250 mg / day, administered after dinner (at 19:00 to 20:00 pm), for 90 days. Design research is quasy experimental pretest-posttest design. Analysis of data using normality test Kolmogorof Smirnof-Z (p>0.05), Levene homogeneity test (p>0.05), t test (p<0.05) and Pearson correlation test (p <0.05). Results: The results showed systolic blood pressure after consume goat milk decreased significantly in the treatment group compared to the control group (122 ± 7:33 and 10:54 ± 115 vs 119 ± 7.61 ± 4.83 mmHg and 118 mmHg; p <0.05), whereas diastolic blood pressure in the treatment group and the control group (80.42 ± 5:53 and 7:08 ± 78.42 mmHg vs; 78.50 ± 3:37 and 3:16 ± 79 mmHg; p> 0.05) did not show differences after administration of goat’s milk. Serum calcium levels after administration of dairy goats in the treatment group increased significantly compared with the control group (9:47 ± 0:25 and 0:32 ± 9.87 mg / dl vs 9.74 ± 0:42 and 9:37 ± 0:38 mg / dl; p <0.05). The results of Pearson correlation test (r) showed r=-0.45; p=0.05, meaning there were nonsignificant correlation between systolic blood pressure with serum calcium levels. Conclusion: Delivery of goat’s milk can decrease systolic blood pressure and stimulates the secretion of calcium, but a decrease in systolic blood pressure was not associated with increased serum calcium levels in people trained.     Abstrak Latar Belakang: Kalsium berperan dalam mengatur tekanan darah dan salah satu sumber kalsium eksogen adalah susu kambing. Masyarakat Indonesia umumnya percaya bahwa susu kambing dapat menurunkan tekanan darah dan bermanfaat sebagai antihipertensi, namun sejauh ini belum ditemukan bukti ilmiah bagaimana kerja susu kambing dalam mengontrol tekanan darah. Penelitian ini bertujuan menganalisis pengaruh konsumsi susu kambing dalam menurunkan tekanan darah dan hubungannya dengan kadar kalsium serum pada orang terlatih. Metode: Subjek, 19 pesenam (kelompok perlakuan) dan 10 atlet lari (kelompok kontrol), laki-laki dan perempuan, usia 17-28 tahun. Perlakuan: pemberian susu kambing 250 mg/hari, diberikan setelah makan malam (pukul 19.00-20.00 wib), selama 90 hari. Design penelitian adalah quasy experimental pretest-posttest design. Analisis data menggunakan uji normalitas Kolmogorof Smirnof-Z (p>0,05), uji homogenitas Levene (p>0,05), uji t (p<0,05) dan uji korelasi pearson (p<0,05). Hasil: hasil penelitian menunjukkan tekanan darah sistolik setelah pemberian susu kambing pada kelompok perlakuan menurun signifikan dibandingkan kelompok kontrol (122±7.33 dan 115±10.54 vs 119±7.61 dan 118±4.83 mmHg mmHg; p<0,05), sedangkan tekanan darah diastolic pada kelompok perlakuan dan kelompok kontrol (80.42±5.53 dan 78.42±7.08 mmHg vs; 78.50±3.37 dan 79±3.16 mmHg; p>0,05) tidak menunjukkan perbedaan setelah pemberian susu kambing. Kadar kalsium serum setelah pemberian susu kambing pada kelompok perlakuan meningkat signifikan dibandingkan dengan kelompok kontrol (9.47±0.25 dan 9.87±0.32 mg/dl vs 9.74±0.42 dan 9.37±0.38 mg/dl; p<0,05). Hasil uji korelasi pearson (r) menunjukkan r=-0,45; p=0,05, artinya terdapat korelasi sedang yang tidak bermakna antara tekanan darah sistolik dengan kadar kalsium serum. Kesimpulan: Pemberian susu kambing dapat menurun tekanan darah sistolik dan merangsang sekresi kalsium namun penurunan tekanan darah sistolik tidak berhubungan dengan peningkatan kadar kalsium serum pada orang terlatih.

Hemodynamic Profiles as a Predictor of Mortality and Length Of Stay in ICCU: Insight from Registry of Acute and Intensive Cardiovascular Care Outcome Firdaus, Isman; Lilihata, Gracia; Kristianto, Ardeno; Simanjuntak, Cindya K.; Danny, Siska S.; Irmalita, Irmalita; Dharma, Surya; Juzar, Dafsah A.; Tobing, Daniel P.L.
Indonesian Journal of Cardiology Vol 38 No 3 (2017): July - September 2017
Publisher : The Indonesian Heart Association

Aims: The ability to differentiate high risk and low risk patients in ICCU is beneficial. Hemodynamic profiles can be used to describe patient’s condition immediately. Based on the presence of congestion and poor perfusion, patients can be divided into four hemodynamic profiles. We aim to evaluate the prognostic value of hemodynamic profiles for patient’s mortality and length of stay (LOS) in intensive cardiac care unit (ICCU). Methods: In this retrospective cohort study, patients who admitted to ICCU of National Cardiovascular Center Harapan Kita Jakarta, Indonesia, were classified into four hemodynamic profiles: dry-warm, dry-cold, wet-warm, and wet-cold. Bivariate analysis was performed to see the significance between hemodinamic profiles with mortality and LOS, continued with multvariate analysis to evaluate the contribution of other significant factors. Results: Of 742 patients included, the mortality rate was 7.8%. With dry-warm profile as reference, relative risk for mortality was 2.3 (95% CI 1.303-4.076), 5.8 (95% CI 1.992-16.906), and 8.7 (95% CI 3.513-21.567) for wet-warm, dry-cold and wet cold, consecutively. Mean differences of LOS (days) as follows: wet-warm (1.719; 95% CI 1.21-2.23), dry-cold (3.418; 95% CI 1.52-5.32), and wet-cold (4.654; 95% CI 2.64-6.67) compared to dry-warm. Hemodynamic profiles, especially wet-cold profile, consistently predicted mortality and longer LOS in ICCU by multivariable analysis. Conclusion: The presence of “wet” profile double the risk of death, “cold” profile has five fold risk of death, while the presence of both has the highest risk for mortality and longer LOS. Hemodynamic profiles assessme   Abstrak Latar Belakang: Kemampuan untuk membedakan pasien resiko tinggi dan resiko rendah di ICCU sangat penting. Profil hemodinamik dapat digunakan untuk mengenali kondisi pasien secara cepat. Berdasarkan adanya tanda kongesti dan perfusi yang buruk pasien dapat dikelompokkan ke dalam empat profil hemodinamik. Studi ini bertujuan untuk mengevaluasi nilai prognostik profil hemodinamik terhadap mortalitas dan lama rawat pasien di Intensive Cardiac Care Unit (ICCU). Metode : Studi kohort retrospektif ini dilakukan di Rumah Sakit Pusat Jantung dan Pembuluh Darah Nasional Harapan Kita, Jakarta, Indonesia. Pasien yang dirawat di ICCU dikelompokkan ke dalam empat profil hemodinamik: kering-hangat, kering-dingin, basah-hangat dan basah-dingin. Analisis bivariate dilakukan untuk menilai hubungan antara profil hemodinamik dengan mortalitas dan lama rawat di ICCU, dilanjutkan dengan analisis multivariate untuk mengevaluasi kontribusi faktor-faktor lain yang signifikan Hasil : Total pasien yang ikut dalam studi sebanyak 742 pasien dan tingkat mortalitas sebesar 7,8%. Resiko relatif (RR) mortalitas untuk profil basah-hangat, kering-dingin dan basah-dingin berturut-turut sebesar 2.3 (95% CI 1.303-4.076), 5.8 (95% CI 1.992-16.906), dan 8.7 (95% CI 3.513-21.567) bila dibandingkan terhadap profil kering-hangat sebagai referensi. Rerata perbedaan lama rawat sebesar 1.719 (95% CI 1.21-2.23), 3.418 ( 95% CI 1.52-5.32), (4.654 (95% CI 2.64-6.67) untuk profil basah-hangat, kering-dingin, dan basah dingin berturut-turut bila dibandingkan dengan profil kering-hangat. Profil hemodinamik, terutama profil basah-dingin secara konsisten memprediksi mortalitas dan lama rawat yang lebih panjang setelah analisis multivariat. Kesimpulan: Profil “basah” memiliki resiko mortalitas dua kali lipat, profil “dingin” memiliki resiko mortalitas lima kali lipat, sedangkan keduanya secara bersamaan memiliki resiko mortalitas dan lama rawat lebih panjang paling tinggi. Profil hemodinamik dapat digunakan sebagai prediktor mortalitas dan lama rawat pasien di ICCU secara efektif.  

Predictor of Left Atrial Spontaneous Echocardiographic Contrast in Rheumatic Mitral Stenosis Patients Komaria, Komaria; Halim R, Abdul; Nst, Ali Nafiah; Hasan, Harris
Indonesian Journal of Cardiology Vol 38 No 3 (2017): July - September 2017
Publisher : The Indonesian Heart Association

Background: Previously conducted researches showed that presence of SEC in the left atrium can constitute a risk factor for thrombus formation. Some previous studies have also reported that in addition to atrial fibrillation and blood stasis in the left atrium, the pathophysiology of left atrial thrombus and SEC occurring in patients with rheumatic mitral stenosis exhibits some other mechanisms, such as autoimmunity, inflammation and increased thrombotic activity. Methods: Cross sectional study was conducted between July 2015 to July 2017 in patient who admitted to Haji Adam Malik Hospital due to rheumatic mitral stenosis. They were divided into two groups according to presence of left atrial SEC. Result: From 104 patients, 52 (mean age 40 ± 11 years; 71,2% women) were in the left atrial SEC-negative group and 52 patients (mean age 40 ± 10 years; 73,1% women) were in the left atrial SEC-positive group. There were no significant differences in the leucocyte, 8,06±1,54 were in the left atrial SEC-negative group and 7,37±1,76 were in the left atrial SEC-positive group. In multivariate analysis, atrial fibrillation (OR = 51,311, 95% CI 3,723 – 707,100, p = 0,003) neutrophil/lymphocyte ratio (OR = 21,641, 95% CI 5,174 – 90,528, p < 0,001), mitral valve area (OR = 14,423, 95% CI 1,665 – 124,908, p = 0,015), and RDW (OR = 5,743, 95% CI 1,349 – 24,445, p = 0,018), These study show that neutrophil/lymphocyte ratio with cut off point of >3,2 had sensitivity, spesificity, positive predictive value, and negative predictive value to predict left atrial SEC is the same 81%, respectively. Conclusion: Atrial fibrillation, neutrophil/lymphocyte ratio, RDW and mitral valve area can predict left atrial spontaneous echocardiographic contrast in rheumatic mitral stenosis patients.   Abstrak Latar Belakang: Penelitian sebelumnya menunjukkan adanya SEC di atrium kiri menjadi faktor risiko untuk pembentukan trombus. Pada pasien stenosis mitral rematik, risiko trombosis dan perkembangan SEC di atrium kiri tinggi. Beberapa penelitian sebelumnya melaporkan bahwa patofisiologi trombus dan SEC di atrium kiri selain fibrilasi atrium dan stasis aliran darah di atrium kiri juga adanya beberapa mekanisme lain seperti respon imun, inflamasi dan peningkatan aktifitas trombotik. Metode: Ini adalah penelitian observasional yang bersifat cross sectional, dilakukan dari Juli 2015 sampai Juli 2017 terhadap pasien stenosis mitral rematik yang datang ke rumah sakit Haji Adam Malik. Pasien dibagi 2 kelompok berdasarkan kehadiran SEC di atrium kiri menurut hasil pemeriksaan ekokardiografi. Hasil: Didapatkan 104 pasien, dimana 52 pasien (usia rata-rata 40 ± 11 tahun, 71% wanita) merupakan kelompok tanpa SEC, dan 52 pasien (usia rata-rata 40 ± 10 tahun, 73% wanita) merupakan kelompok dengan SEC. Tidak ada perbedaan bermakna pada lekosit, dimana kelompok tanpa SEC (8,06±1,54) dan kelompok dengan SEC (7,37±1,76). Dari analisis multivariat regresi logistik, didapatkan fibrilasi atrium (OR = 51,311, nilai IK 95% antara 3,723 – 707,100, p = 0,003) rasio netrofil/limfosit (OR = 21,641, nilai IK 95% antara 5,174 – 90,528, nilai p < 0,001), area katup mitral (OR = 14,423, nilai IK 95% antara 1,665 – 124,908, nilai p = 0,015), dan RDW (OR = 5,743, nilai IK 95% antara 1,349 – 24,445, nilai p = 0,018), merupakan prediktor independen untuk terjadinya SEC. Titik potong untuk nilai rasio N/L > 3,2 memiliki angka sensitivitas, sensitifitas, nilai prediktif positif dan nilai prediktif negatif yang sama yaitu masing-masing 81% untuk memprediksi kejadian SEC di atrium kiri pada pasien stenosis mitral rematik. Kesimpulan: Fibrilasi atrium, rasio netrofil/limfosit, RDW dan area katup mitral dapat menjadi prediktor SEC di atrium kiri pada pasien stenosis mitral rematik.

A Case Report of Elderly Woman with Supraventricular Tachycardia associated with Intracranial Bleeding Pranata, Raymond; Chintya, Veresa; Yonas, Emir; Damay, Vito
Indonesian Journal of Cardiology Vol 38 No 3 (2017): July - September 2017
Publisher : The Indonesian Heart Association

Background The neurogenic cardiac injury is related to brain injury-induced catecholamine and neuro-inflammatory responses and is more likely in those with the most severe neurological insult. Case Report A 78 years-old female presented to the emergency department after being found lying on the floor with a laceration on the head. On physical examination GCS 3, BP 140/90 mmHg, HR 190 bpm, respiratory rate 25x/minute. PMH of hypertension and diabetes were denied. ECG showed supraventricular tachycardia of 186 bpm. Laboratory exams showed hyponatremia, hypokalemia, and leukocytosis. CT scan revealed subarachnoid hemorrhage, intracerebral hematomas, chronic subdural hematoma, midline shift and subfalcine herniation. The systemic catecholamine ‘storm’ driven by the central neuroendocrine axis massively increases sympathetic outflow, activates the adrenal gland and may lead to arrhythmia. Increased ICP, Midline shift, and subsequent physical compression of the brainstem and hypothalamic autonomic centers can trigger catecholamine responses that could instigate an arrhythmia. Suboptimal cardiac output and cerebral perfusion worsen secondary brain injury leading to a worse prognosis. Since cardioversion failed, amiodarone was administered. Cardioversions failed to convert to sinus rhythm and amiodarone was administered. Therapy to reduce intracranial pressure was also administered. The patient passed away 4 hours after admission. Conclusion Arrhythmia related to brain injury may lead to suboptimal cerebral perfusion and leads to further autonomic derangements leading to a vicious cycle of cerebral and cardiovascular injuries. This condition should be accounted for swiftly to prevent secondary brain injuries and myocardial ischemia.   Abstrak Latar Belakang Respons katekolamin dan radang terhadap cedera otak menyebabkan cedera jantung neurogenik yang lebih sering terjadi pada kerusakan neurologis yang berat. Laporan Kasus Seorang perempuan 78 tahun datang ke IGD setelah ditemukan tergeletak di lantai dengan robek pada kulit kepala. Pada pemeriksaan fisik ditemukan GCS 3, TD 140/90 mmHg, detak jantung 190 kali/menit, laju pernafasan 25 kali/menit. Riwayat hipertensi dan diabetes disangkal. EKG menunjukan takikardia supraventrikular takikardi 186 kali/menit. Pemeriksaan laboratorium menunjukan hiponatremia, hipokalemia dan leukositosis. CT scan menunjukan perdarahan subaraknoid, perdarahan intraserebral, perdarahan subdural kronis, midline shift dan herniasi subfalcine. Badai katekolamin sistemik yang dicetuskan oleh aksis neuroendokrin pusat meningkatkan outflow simpatetik yang mengaktifkan kelenjar adrenal dan menyebabkan aritmia. Peningkatan tekanan intrakranial, midline shift, dan penekanan batang otak serta pusat autonomik hipotalamus dapat mencetuskan respon katekolamin yang dapat menyebabkan aritmia. Curah jantung yang tidak optimal dan perfusi otak yang buruk menyebabkan cedera otak sekunder yang mengarah pada prognosis yang buruk. Karena kardioversi gagal merubah irama menjadi sinus maka amiodaron diberikan. Pengobatan untuk menurunkan tekanan intrakranial juga diberikan. Pasien meninggal 4 jam setelah masuk rumah sakit. Kesimpulan Aritmia yang berhubungan dengan cedera otak dapat menyebabkan perfusi otak yang suboptimal serta menyebabkan ketidakteraturan otonom dan menyebabkan lingkaran setan kerusakan otak dan jantung-pembuluh darah. Hal ini harus segera ditangani untuk mencegah cedera otak sekunder dan iskemi miokardial.

Perioperative Antithrombotic Management Andrianto, Andrianto; Arindanie, Amelia
Indonesian Journal of Cardiology Vol 38 No 3 (2017): July - September 2017
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (0.036 KB)

The management of antithrombotic therapy in the perioperative setting is a common problem, balancing haemorrhagic risk with thrombotic risk. High-quality evidence is lacking regarding the optimal approach for patients on oral anticoagulants or antiplatelet agents. The two main issues that need to be considered in perioperative antithrombotic management is the patient’s risk of a thromboembolic event when therapy is interrupted and the risk of bleeding associated with the surgery or procedure. An assessment of these factors will determine the optimal perioperative anticoagulant management approach. The overall objective of this review is to provide a practical approach relating to perioperative management which can be used in everyday clinical practice.   Abstrak Masalah tatalaksana terapi antitrombotik perioperatif yaitu menyeimbangkan risiko perdarahan dan risiko trombosis masih sering dijumpai sehari-hari. Bukti klinis yang kuat mengenai tatalaksana yang optimal dari terapi antikoagulan atau antiplatelet masih sangat sedikit. Dua hal yang perlu dipikirkan dalam tatalaksana terapi antitrombotik perioperatif adalah risiko kejadian tromboemboli ketika terapi antitrombotik dihentikan dan risiko perdarahan akibat tindakan pembedahan yang dilakukan. Penilaian faktor-faktor tersebut akan menentukan pendekatan yang optimal dari tatalaksana antikoagulan perioperatif. Tinjauan pustaka ini bertujuan memberikan pendekatan praktis tentang tatalaksana antitrombotik perioperatif yang dapat dipakai dalam praktik klinis sehari-hari.

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All Issue Vol 44 No Suppl_A (2023): Abstracts of the 32nd Annual Scientific Meeting of the Indonesian Heart As Vol 44 No Suppl_B (2023): Abstracts of the 10th Annual Scientific Meeting of the Indonesian Heart Rh Vol 43 No 4 (2022): Indonesian Journal of Cardiology: October - December 2022 Vol 43 No 3 (2022): Indonesian Journal of Cardiology: July - September 2022 Vol 43 No 2 (2022): Indonesian Journal of Cardiology: April - June 2022 Vol 43 No Suppl_B (2022): Abstracts of the 6th InaPrevent (2022) Vol 43 No 1 (2022): Indonesian Journal of Cardiology: January - March 2022 Vol 43 No Suppl_C (2022): Abstracts of the 31st Annual Scientific Meeting of the Indonesian Heart As Vol 43 No Suppl_D (2022): Abstracts of the 9th Annual Scientific Meeting of the Indonesian Heart Rhy Vol 43 No Supplement (2022): Abstracts of the 5th Indonesian Intensive and Acute Cardiovascular Care Vol 42 No 4 (2021): Indonesian Journal of Cardiology: October - December 2021 Vol 42 No 3 (2021): Indonesian Journal of Cardiology: July - September 2021 Vol 42 No 2 (2021): Indonesian Journal of Cardiology: April - June 2021 Vol 42 No 1 (2021): Indonesian Journal of Cardiology: January - March 2021 Vol 42 No Supplement (2021): Abstracts of the 5th InaPrevent in Conjunction with the 1st InTension S Vol 42 No Supplement (2021): Abstracts of the 13th Indonesian Society of Interventional Cardiology A Vol 42 No Supplement (2021): Abstracts of the 8th Annual Scientific Meeting of the Indonesian Heart Vol 41 No 4 (2020): Indonesian Journal of Cardiology: October - December 2020 Vol 41 No 3 (2020): Indonesian Journal of Cardiology: July - September 2020 Vol 41 No 2 (2020): Indonesian Journal of Cardiology: April - June 2020 Vol 41 No 1 (2020): Indonesian Journal of Cardiology: Januari - Maret 2020 Vol 41 No Suppl_A (2020): Abstracts of the 29th Annual Scientific Meeting of the Indonesian Heart As Vol 41 No Suppl_B (2020): Abstracts of the 12th Indonesian Society of Interventional Cardiology Annu Vol 40 No 4 (2019): Indonesian Journal of Cardiology: October-December 2019 Vol 40 No 3 (2019): Indonesian Journal of Cardiology: July-September 2019 Vol 40 No 2 (2019): Indonesian Journal of Cardiology: April-June 2019 Vol 40 No 1 (2019): Indonesian Journal of Cardiology: January-March 2019 Vol 39 No 4 (2018): Indonesian Journal of Cardiology: October-December 2018 Vol 39 No 3 (2018): Indonesian Journal of Cardiology: July-September 2018 Vol 39 No 2 (2018): Indonesian Journal of Cardiology: April-June 2018 Vol 39 No 1 (2018): January - March 2018 Vol 39 No 1 (2018): January - March 2018 Vol 39 No Suppl_B (2018): Indonesian Journal of Cardiology Supplement_B Vol 39 No Suppl_B (2018): Indonesian Journal of Cardiology Supplement_B Vol 39 No Suppl_A (2018): Indonesian Journal of Cardiology Supplement_A Vol 39 No Suppl_A (2018): Indonesian Journal of Cardiology Supplement_A Vol 38 No 4 (2017): October - December 2017 Vol 38 No 4 (2017): October - December 2017 Vol 38 No 3 (2017): July - September 2017 Vol 38 No 3 (2017): July - September 2017 Vol. 38, No. 2 April-June 2017 Vol. 38, No. 2 April-June 2017 Vol. 38, No. 1 Januari-Maret 2017 Vol. 37, No. 4 Oktober - Desember 2016 Vol. 37, No. 3 Juli - September 2016 Vol. 37, No. 2 April - Juni 2016 Vol. 37, No. 1 Januari - Maret 2016 Vol. 36, No. 4 Oktober - Desember 2015 Vol. 36, No. 3 Juli - September 2015 Vol. 36, No. 2 April - Juni 2015 Vol. 36, No. 1 Januari - Maret 2015 Vol. 35, No. 4 Oktober - Desember 2014 Vol. 35, No. 3 Juli - September 2014 Vol. 35, No. 2 April - Juni 2014 Vol. 35, No. 1 Januari - Maret 2014 Vol. 34, No. 4 Oktober - Desember 2013 Vol. 34, No. 3 Juli - September 2013 Vol. 34, No. 2 April - Juni 2013 Vol. 34, No. 1 Januari - Maret 2013 Vol. 33, No. 4 Oktober - Desember 2012 Vol. 33, No. 3 Juli - September 2012 Vol. 33, No. 2 April - Juni 2012 Vol. 33, No. 1 Januari - Maret 2012 Vol. 32, No. 4 Oktober - Desember 2011 Vol. 32, No. 3 Juli - September 2011 Vol. 32, No. 2 April - Juni 2011 Vol. 32, No. 1 Januari - Maret 2011 Vol. 31, No. 3 September - Desember 2010 Vol. 31, No. 2 Mei - Agustus 2010 Vol. 31, No. 1 Januari - April 2010 Vol. 30, No. 3 September - Desember 2009 Vol. 30, No. 2 Mei - Agustus 2009 Vol. 30, No. 1 Januari - April 2009 Vol. 29, No. 3 September - Desember 2008 Vol. 29, No. 2 Mei - Agustus 2008 Vol. 29, No. 1 Januari - April 2008 Vol. 28, No. 6 November 2007 Vol. 28, No. 5 September 2007 Vol. 28, No. 4 Juli 2007 Vol. 28, No. 3 Mei 2007 Vol. 28, No. 2 Maret 2007 Vol. 28, No. 1 Januari 2007 Vol. 26, No. 1 Januari - Maret 2002 More Issue

Home Page

OAI Link

Editorial Team

Contact

Reviewer

Google Scholar

Contact Name
Raymond Pranata

Contact Email
raymond_pranata@hotmail.com

Phone
+6282112918892

Journal Mail Official
ijc@inaheart.org

Editorial Address
Editorial Office: Heart House, Jalan Katalia Raya No. 5, Kota Bambu Utara West Jakarta, 11430 - Indonesia Telephone: +62 21 5681149, Fax: +62 21 5684220 Email: ijc@inaheart.org

Location
Kota adm. jakarta barat,

Dki jakarta

INDONESIA

Abstract

Abstract

Abstract

Abstract

Abstract

Abstract

Abstract

Abstract

Abstract

Abstract

Home Page

OAI Link

Editorial Team

Contact

Reviewer

Google Scholar

Contact Name Raymond Pranata

Contact Email raymond_pranata@hotmail.com

Phone +6282112918892

Journal Mail Official ijc@inaheart.org

Editorial Address Editorial Office: Heart House, Jalan Katalia Raya No. 5, Kota Bambu Utara West Jakarta, 11430 - Indonesia Telephone: +62 21 5681149, Fax: +62 21 5684220 Email: ijc@inaheart.org

Location Kota adm. jakarta barat, Dki jakarta INDONESIA

Abstract

Abstract

Abstract

Abstract

Abstract

Abstract

Abstract

Abstract

Abstract

Abstract

Contact Name
Raymond Pranata

Contact Email
raymond_pranata@hotmail.com

Phone
+6282112918892

Journal Mail Official
ijc@inaheart.org

Editorial Address
Editorial Office: Heart House, Jalan Katalia Raya No. 5, Kota Bambu Utara West Jakarta, 11430 - Indonesia Telephone: +62 21 5681149, Fax: +62 21 5684220 Email: ijc@inaheart.org

Location
Kota adm. jakarta barat,

Dki jakarta

INDONESIA