cover
Article Per Year (5 Year)
All
Home Page
OAI Link
Editorial Team
Contact
Reviewer
Google Scholar
Contact Name
Nyoman Angga Krishna Pramana
Contact Email
anggakrishna@gmail.com
Phone
-
Journal Mail Official
anggakrishna.dr@gmail.com
Editorial Address
-
Location
Unknown,
Unknown
INDONESIA
Callosum Neurology Journal : Jurnal Berkala Neurologi Bali
Published by Perdossi Cabang Denpasar
ISSN : 26140276     EISSN : 26140284     DOI : -
Core Subject : Health, Science,
Health Professions Medicine & Pharmacology Neuroscience
Callosum Neurology Journal is an official journal managed by The Indonesia Neurological Association XXV Branch of Denpasar. This journal is open access to the rules of peer-reviewed journaling which aims as scientific publications and sources of actual information in the field of neurology and neuroscience. Callosum Neurology Journal is published three times a year in January, May, and September and contains original articles of research, case reports, case series reports, literature reviews, and communications from and to editors.
Arjuna Subject : -
Articles 75 Documents
 4   5   6   7   8 
MONOPARESIS INFERIOR POST HERPES ZOSTER Pande Komang Novi Dyantari; I Ketut Sumada
Callosum Neurology Vol 4 No 2 (2021): Callosum Neurology Journal
Publisher : The Indonesia Neurological Association Branch of Denpasar

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.29342/cnj.v4i2.149

Abstract

Background: Herpes zoster (HZ) is caused by reactivation of Varicella Zoster Virus (VZV). Segmental zoster paresis (SZP) is a rare neurological complication of HZ. Case: A 58-year-old woman came to our clinic with a complaint of a post HZ lesion accompanied by weakness, burning sensation, and paresthesia of the left lower limb. These complaints were felt suddenly about 2 weeks after the vesicles appeared. The patient had a history of diabetes mellitus (DM) and hypertension. From physical examination, there was a lesions on the L2-L3 dermatome which had dried out with hyperpigmentation and crusted.  Motoric examination of the left lower limb showed weakness of the hip flexors (MRC grade 3/5). While the power of the other muscles in right lower limb and in all muscles groups was MRC 5/5. Tonus and trophic was normal. There was hyperaesthesia noted in the left thigh in the L2-L3 dermatome. Discussion: Neurological complications in HZ can occur in the central nervous system or peripheral nervous system. In this case the patient had SZP as a complication of HZ that involving the motor system. This complication was characterized by paresis of the left lower limb after developing HZ infection. The pathogenesis of SZP remains unclear. Having a history of DM increases the incidence of neurological complications in HZ. This is because the effectiveness of cellular immunity against VZV in DM patients decreases. Conclusion: Segmental zoster paresis (SZP) is a rare neurological complication of HZ. SZP involves the motor nervous system. There are several risk factors that increase the incidence of neurological complications in HZ infection. Key words: Herpes Zoster, Segmental Zoster Paresis  
GEJALA PSIKOTIK PADA PENYAKIT PARKINSON: SEBUAH LAPORAN KASUS DAN TINJAUAN PUSTAKA Sri Yenni Trisnawati; Purwa Samatra Dewa Putu Gde; Lely Setyawati Kurniawan; Astari Arum Cendani Goller; Valentina Tjandra Dewi
Callosum Neurology Vol 4 No 2 (2021): Callosum Neurology Journal
Publisher : The Indonesia Neurological Association Branch of Denpasar

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.29342/cnj.v4i2.152

Abstract

Pendahuluan: Penyakit Parkinson (PP) memiliki dimensi gejala sangat luas yang secara langsung dan tidak langsung mempengaruhi kualitas hidup penderita maupun keluarga. Gejala non motorik PP diantaranya adalah gangguan neuropsikiatri seperti depresi, psikosis, ansietas. Kami melaporkan sebuah kasus PP dengan gejala psikotik berupa halusinasi yang jarang dijumpai. Laporan Kasus: Pasien perempuan berusia 74 tahun terdiagnosis Parkinson sejak lebih dari 20 tahun, dibawa ke rumah sakit dengan bicara tidak nyambung, adanya halusinasi visual, dan emosi yang tidak stabil sejak 1 bulan. Keluarga pasien juga merasa sejak 6 bulan terakhir pasien menjadi sering lupa. Pasien dengan riwayat PP tidak rutin mengkonsumsi obat parkinson di bawah supervisi dokter. Keluhan halusinasi pernah dirasakan 2 tahun lalu namun membaik setelah perawatan. Diskusi: Gejala non motorik dapat dijumpai seiring perkembangan penyakit Parkinson dandapat diidentifikasi sebelum sindrom motor muncul. Adanya gejala psikosis berupa halusinasi dalam kasus yang dipaparkan terhadi setelah 18 tahun sejak pasien terdiagnosis, dimana berdasarkan hasil studi terdahulu dilaporkan 60% penderita PP berkembang adanya halusinasi atau delusi setelah 12 tahun perawatan dan pada usia lebih tua saat onset PP. Gangguan kognitif pada PP juga dicurigai dalam kasus walau belum sepenuhnya dapat ditegakan serta adanya gangguan tidur pada PP dengan prevalensi 40-90% dijumpai pada kasus yang kami laporkan. Kesimpulan: Manifestasi non motorik PP yang mencakup gejala neuropsikiatri sangat mempengaruhi kualitas hidup pasien maupun keluarga dan perawat pasien. Klinisi harus meningkatkan kewaspadaan terhadap gejala non motorik PP dan pemberian tatalaksana farmakologi dan non farmakologi harus dilakukan secara terintegratif bersama dengan bidang-bidang terkait sesuai dengan kondisi pasien. Kata kunci: Parkinson, non-motor, psikotik, neuropsikiatri
LESI NERVUS OKULOMOTOR INVOLVED PUPIL ET CAUSA ANEURISMA PADA PASIEN DENGAN STROKE SUBARACHNOID HEMORRHAGIC DAN SYSTEMIC LUPUS ERITHEMATOSUS I Ketut Aryawan; Anak Agung Mas Putrawati Triningrat; Made Paramita Wijayanti; Ida Ayu Sri Indrayani; Pande Ketut Kurniari
Callosum Neurology Vol 4 No 2 (2021): Callosum Neurology Journal
Publisher : The Indonesia Neurological Association Branch of Denpasar

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.29342/cnj.v4i2.163

Abstract

Introduction: Oculomotor nerve palsy is an pathological condition caused by microvascular injury, head injury, compression due to neoplasm or aneurysm, and also oculomotor nerve palsy can be caused by autoimmune prosses. Peripheral neuropathy is one of the clinical manifestations in patient with SLE, Oculomotor nerve palsy is one type of cranial neuropathy seen with SLE patient. Patient with SLE have a higher risk of serebrovascular event than general population. Case Illustration: Female 34 years old complained drop of the eyelid on the left eye and double vision when see with both eyes since 1 mounth before examination. Patient with history of headace and diagnosed with SLE since 2006 with regular treatment. From the examination, pupil anisocor, on the right eye pupil was 3 mm in diameter with positif direct and indirect reflex. On the left eye pupil was 6 mm with negative direct and indirect reflex. Extraocular movement on the left eye was limited except abduction movement.  CT-Scan examination shows bilateral subarachnoid hemorrhage and from CT-Angiography shows dilatation of the left siphon carotid artery. Patient was diagnosed with oculomotor nerve palsy involving pupil caused by aneurysm with SAH and SLE. Discussion: Oculomotor nerve palsy mostly caused by aneurysm compression in posterior communicating artery (PCoA) and internal carotid artery (ICA). Cerebrovascular imaging, MRA and CTAngiography, can showing the aneurysm and its location. Risk of cerebrovascular event increased in patient with SLE than general population. Management patient with oculomotor nerve palsy with SLE nowadays is with pulse dose corticosteroid. Conclusion: SLE with oculomotor nerve palsy will increase risk of cerebrovascular event. Key Words : Oulomotor Nerve Palsy, Subarachnoid Hemorrhage, Systemic Lupus Erithematosus
PENINGKATAN D-DIMER DAN LUARAN KLINIS PASIEN TERKONFIRMASI COVID-19 YANG MENGALAMI STROKE DI RSUP SANGLAH : LAPORAN KASUS Ida Ayu Sri Indrayani; Andre Dharmawan Wijono; Aurelia Vania; Edwin Pranata Laban; Johan Andrasilli; Muhammad Arismunandar; Fuji Restu Firma
Callosum Neurology Vol 4 No 2 (2021): Callosum Neurology Journal
Publisher : The Indonesia Neurological Association Branch of Denpasar

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.29342/cnj.v4i2.164

Abstract

Latar Belakang: COVID-19 sering dihubungkan dengan terjadinya hiperkoabilitas yang menyebabkan komplikasi pada sistem neurovascular, salah satunya adalah stroke. D-dimer merupakan salah satu pemeriksaan yang dapat digunakan untuk melihat adanya koagulopati. Tinjauan ini bertujuan untuk melihat pentingnya pemeriksaan d-dimer pada pasien COVID-19 yang mengalami stroke. Kasus: Dari data yang diambil di RSUP Sanglah bulan Juni sampai dengan Agustus 2020, didapatkan 4 pasien stroke non hemoragik yang terkonfirmasi COVID-19. Keempat pasien tersebut berusia 80, 75, 59, dan 76 tahun dimana semua pasien berjenis kelamin laki-laki. Klinis neurologis yang didapatkan pada keempat pasien tersebut yaitu paresis nervus cranialis VII, paresis nervus cranialis XII dan hemiparesis flaksid disertai dengan adanya refleks patologis. Dari keempat pasien tersebut, hanya ada satu pasien yang tidak mengalami sesak napas. Tiga pasien mengalami pneumonia pada pemeriksaan rontgen thorax. Setelah dilakukan pemeriksaan d-dimer, semua pasien menunjukkan peningkatan angka D-dimer dengan angka masing-masing 0.73, 1.03, 7.03, dan 1.51. Pasien dengan D-dimer 7.03 memiliki skor NIHSS tertinggi yaitu 8. Diskusi: Proses apoptosis sel-sel endotel dari struktur vaskular mengakibatkan terjadinya koagulopati dan peningkatan D-dimer. Studi melaporkan hasil laboratorium D-dimer yang lebih tinggi pada kondisi COVID-19 berat atau COVID-19 dengan gangguan serebrovaskular. Mekanisme gangguan serebrovaskular tanpa faktor risiko vaskular sebelumnya diduga berasal dari kondisi hiperkoagulasi yang menyebabkan pembentukan trombus dalam pembuluh darah. D-dimer meningkat pada 36% pasien dengan COVID-19 di Wuhan, yang dikaitkan dengan risiko kematian yang lebih tinggi. Kesimpulan: Kadar D-dimer berkorelasi dengan keparahan penyakit dan merupakan suatu penanda prognostik tingkat keparahan pada pasien yang dirawat karena COVID-19. Kata kunci: D-dimer, Covid-19, stroke, luaran klinis
COVID-19 AS A POTENTIAL RISK FACTOR FOR ISCHEMIC STROKE: A CASE REPORT Orlando Pikatan; Desie Yuliani; I Ketut Sumada; Ni Ketut Candra Wiratmi
Callosum Neurology Vol 4 No 2 (2021): Callosum Neurology Journal
Publisher : The Indonesia Neurological Association Branch of Denpasar

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.29342/cnj.v4i2.168

Abstract

Background: Corona virus disease 2019 (COVID-19) is a pandemic disease with a wide spectrum of symptoms.  Neurological symptoms are often found in this disease. Stroke on the other hand is a multifactorial disease that can be caused by a lot of underlying problems. Case: We report a case of a 67 years old man with a weakness on the right side that was worsened one day before arrival in hospital and a shortness of breath. the patient had history of stroke before, and controlled hypertension. Head Ct-scan showed a stroke infarct, and Swab RT-PCR showed + SARS-CoV-2. Discussion: Infection may become the risk factor of stroke. inflammatory mediators such as TNF-a , and CRP  support the procoagulant state which leads to stroke. Conclusion: COVID-19 may increase the risk of stroke due to the inflammation state that leads to procoagulant state  

Page 8 of 8 | Total Record : 75

 4   5   6   7   8 

Filter by Year

2018 2021


Reset
Filter By Issues
All Issue Vol 4 No 2 (2021): Callosum Neurology Journal Vol 4 No 1 (2021): Callosum Neurology Journal Vol 3 No 3 (2020): Callosum Neurology Journal Vol 3 No 2 (2020): Callosum Neurology Journal Vol 3 No 1 (2020): Callosum Neurology Journal Vol 2 No 3 (2019): Callosum Neurology Journal Vol 2 No 2 (2019): Callosum Neurology Journal Vol 2 No 1 (2019): Callosum Neurology Journal Vol 1 No 3 (2018): Callosum Neurology Journal Vol 1 No 2 (2018): Callosum Neurology Journal Vol 1 No 1 (2018): Callosum Neurology Journal More Issue