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Anxiety And Risk Of Cardiovascular Disease Komang Rama Agastya; Andi Agung Riatmojo; Rauchsan Abdi Akbar
Jurnal KESANS : Kesehatan dan Sains Vol 1 No 2 (2021): KESANS : International Journal of Health and Science
Publisher : Rifa'Institute

Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (218.189 KB) | DOI: 10.54543/kesans.v1i2.13

Abstract

Anxiety is a normal emotion that is owned by every individual. Pathologically increased anxiety can not only give rise to other anxiety disorders but can also lead to other mental illnesses. Anxiety is a type of mental disorder that is quite common in Europe, Switzerland, Iceland, Norway in 2010 it was found that the prevalence for 12 months was 14% and 61.5 million people were affected. According to WHO in the global population with anxiety disorders in 2015 the percentage was 3.6% and it was more common in women with a percentage of 4.6% compared to 2.6% for men. Individuals with symptoms of anxiety are likely to experience the effects of interference that can lead to metabolic syndrome. Metabolic syndrome leads to progressive and clustering of several metabolic risk factors that directly increase the risk of death from cardiovascular disease (CVD, type 2 diabetes mellitus). last year from 10% to 63.2%. Anxiety disorders appear to be caused by the interaction of bio-psychosocial factors. Genetic susceptibility interacts with stressful situations or the presence of trauma to produce a clinically significant syndrome. Treatment measures include pharmacological, psychological, and combination therapies. all are therapeutic measures for all anxiety disorders, selective serotonin is a first line agent for several anxiety disorders, including panic disorder, social anxiety, generalized anxiety disorder.Psychotherapy and pharmacotherapy should be given and both therapies are equally effective. The choice in determining the therapy given must be considered according to the severity of the disorder.
Hubungan Variasi Genetik Gen FADS pada Suku Sasak Terhadap Angka Kecukupan Gizi Ridha Sasmitha Ajiningrum; Luh Gde Sri Adnyani; Andi Agung Riatmojo; Vira Eka Trie Sanggita; Siti Rahmah
Jurnal Syntax Fusion Vol 2 No 01 (2022): Jurnal Syntax Fusion: Jurnal Nasional Indonesia
Publisher : Rifa' Institute

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.54543/fusion.v2i01.137

Abstract

One of the macronutrients needed by the human body is fat. Fatty acids are divided into two, namely saturated and unsaturated fatty acids. Based on the source of unsaturated fatty acids are divided into two, namely non-essential and essential. Essential fatty acids consist of omega-3 and omega-6 which are part of PUFAs. To determine the relationship of genetic variation of the FADS gene to the nutritional adequacy of long-chain polyunsaturated fatty acids (LC-PUFA) in children of the Sasak ethnic. Systematic reviews were carried out through scientific journals, government guidelines, online portals for journal publications such as Medscape, Google Scholar, PubMed, and Science Direct, with the keyword “Polyunsaturated Fatty Acid (PUFA)” There was a significant relationship between the FADS2 index (DHA: EPA ratio) and MDI score (r = 0.648, p < 0.059) in AG but not in GG genotype. Two genotype models were found, namely homozygous major allele (GG) of rs 174468 which was dominant and heterozygous allele (AG) of Sasak ethnicity. The levels of eicosapentaenoic acid (EPA) and docosahexaenoic acid (DHA) in infants in the Sasak ethnic were found to be higher than infants in other areas. Sasak ethnic children get EPA and DHA from breast milk which contains a lot of DHA. Sources of omega-3 and 6 on the island of Lombok are sea fish, seaweed, and full-blooded shellfish.
Penurunan Fungsi Sistem Penghidu Pada Pasien Penyakit Parkinson: Epidemiologi dan Tatalaksananya Andi Agung Riatmojo
Jurnal Syntax Fusion Vol 2 No 01 (2022): Jurnal Syntax Fusion: Jurnal Nasional Indonesia
Publisher : Rifa' Institute

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.54543/fusion.v2i01.144

Abstract

Parkinson's disease in general is a disorder of the central nervous system which most of its manifestations are motor function disorders. However, today's non-motor symptoms are found as early manifestations of Parkinson's disease. To determine the relationship between Parkinson's disease and non-motor symptoms, namely loss of smell or anosmia. The writing of this article includes various sources originating from scientific journals on online portals of journal publications such as MedScape, Google Scholar, PubMed, and NCBI, with the keywords "olfactory disorders and Parkinson's disease". Loss of smell can be the first symptom of pathological abnormalities in the central nervous system (CNS). Several studies related to Parkinson's disease stated that almost all patients studied had atrophy of the olfactory bulb, causing the patient to experience loss of smell function (anosmia). The effect of decreasing the volume of the olfactory bulb and the effect of neurotransmitters are also believed to affect the olfactory dysfunction in patients with Parkinson's disease. Anosmia is the most common non-motor symptom in Parkinson's disease, and often precedes motor symptoms in Parkinson's disease. Olfactory training is a promising modality for the treatment of olfactory dysfunction and anosmia. The results of this systematic review and meta-analysis suggest that it may be an effective treatment for olfactory dysfunction due to various etiologies