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Aulia Dwi Hendriani
Universitas Mataram
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Comparison of Bone Conduction of Hearing in Patients with Benign and Malignant Types of Chronic Suppurative Otitis Media Aulia Dwi Hendriani; M. Andre Darmawan; Sima Smith; Ni Kadek Mega Suryantini; Theophany Margareta Kurniawan
Experimental Student Experiences Vol. 1 No. 8 (2023): August
Publisher : LPPM Sunan Doe

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.58330/ese.v1i8.336

Abstract

One of the main problems associated with hearing loss is chronic suppurative otitis media (CSOM). The purpose of this study was to determine the comparison of conduction of the auditory ossicles in patients with benign type chronic suppurative otitis media and malignant type otitis. Chronic suppurative media (CSOM) is a chronic infection of the mucosa and periosteum of the middle ear which is characterized by continuous discharge of fluid from the ear canal and perforation of the tympanic membrane. CSOM is divided into two types, namely the safe type or what is called benign and the dangerous or malignant type. Patients suffering from CSOM can experience conductive hearing loss which has a fairly high incidence rate. Chronic suppurative otitis media is one of the most common diseases in the world, especially in developing countries, which if not treated quickly can lead to seriousness. So the researchers wanted to examine the comparison of hearing bone conduction in patients with benign and malignant type of chronic suppurative otitis media. In addition, there are significant differences in the distribution of types of hearing loss between benign and malignant types of Chronic Suppurative Otitis Media (CSOM) patients. The benign type of CSOM is dominated by conductive deafness, while the malignant type of CSOM tends to have conductive and mixed deafness.
Liver Cirrhosis: Pathophysiology, Diagnosis, and Management Baiq Nadya Putri Maharani; Aulia Dwi Hendriani; Putu Wika Pramesti Iswari
Jurnal Biologi Tropis Vol. 23 No. 1 (2023): Special Issue
Publisher : Biology Education Study Program, Faculty of Teacher Training and Education, University of Mataram, Indonesia

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.29303/jbt.v23i1.5763

Abstract

Liver disease is still a concern in world health and liver cirrhosis is the eleventh leading cause of death in the world. Cirrhosis caused 1.32 million deaths in 2017. Liver cirrhosis is a fibrosis or nodule formation in the liver. The study was conducted on databases, such as PubMed, google scholar and gray literature. With inclusion criteria, that are free full text publications published in 2015-2022 and having relevant discussions. Fibrosis in cirrhosis of liver begins with the activation of Stellate and Kupffer cells, damaged hepatocytes and activated platelets are also invoved. Inflammatory cells will appear as a result of the damage and cause fibrosis due to the secretion of cytokines. In addition, there are pathological features, namely nodule regeneration and loss of normal lobular architecture within the nodule. The patient's diagnosis start from anamnesis to gather information related to risk factors, physical examination, laboratory tests, imaging, liver biopsy if necessary. Management can be carried out according from etiology of the cause of liver cirrhosis. Complications of liver cirrhosis are portal hypertension, ascites, hepatic encephalopathy, hepatocellular carcinoma, hyponatremia, and acute kidney injury. Liver cirrhosis is a liver fibrosis caused by Stellate, Kupffer cells, damaged hepatocytes, and activated platelets. Inflammatory cells cause fibrosis, leading to regenerating nodules and decreased blood flow. Diagnosis involves anamnesis, physical examination, laboratory tests, imaging, and liver biopsy if needed. Treatment is based on the etiology of liver cirrhosis, with complications including portal hypertension, ascites, and hepatic encephalopathy.
Co-Authors Baiq Nadya Putri Maharani M. Andre Darmawan Ni Kadek Mega Suryantini Putu Wika Pramesti Iswari Sima Smith Theophany Margareta Kurniawan