Articles
<![CDATA[KEJADIAN HIV/AIDS DENGAN MANIFESTASI ORAL PADA ANAK-ANAK ]]>
<![CDATA[Hikmah, Nuzulul]]>;
<![CDATA[Dewanti, I Dewa Ayu Ratna]]>
<![CDATA[ STOMATOGNATIC- Jurnal Kedokteran Gigi Vol 7, No 1 (2010) ]]>
Publisher : <![CDATA[Fakultas Kedokteran Gigi Universitas Jember ]]>
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<![CDATA[Nowadays HIV/AIDS infection was increase to children in the world. In development countries include Indonesia, it was caused fourth killer to children. Oral manifestation to the children who has infected HIV have a different been compared with adult. Discussion.Candidiasis and necrotizing ulcerative gingivitis is oral lesion who was found in the children. Linear gingival erythema leukoplakia, caries and kaposis sarcoma can be an indicator of moderate and hard immunosupression. Parotis swollen, herpes simplex virus infection and recurrent aphtous stomatitis not special connected with immunosupression grade. The best of management patients was consider immunology status andexamined oral continuity. Conclusion. Oral manifestation in the childen very important to known because it was often first indication clinical so that someone had been infected of HIV.]]>
<![CDATA[INFEKSI Cytomegalo virus (CMV) PADA IBU HAMIL DAN MANIFESTASINYA DI RONGGA MULUT ]]>
<![CDATA[Dewanti, I Dewa Ayu Ratna]]>
<![CDATA[ STOMATOGNATIC- Jurnal Kedokteran Gigi Vol 7, No 1 (2010) ]]>
Publisher : <![CDATA[Fakultas Kedokteran Gigi Universitas Jember ]]>
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<![CDATA[Immune status in pregnancy was very impotant, because the effect for life to fetus andmother. If immune in pregnancy hasânt been attended, it will attack infection. Infection diseases was found in pregnancy was Cytomegalo virus (CMV) infection. Aims. This literature reviews purposes to information of CMV infection in pregnancy and manifestation in oral mucosa. Discussion. Infection in fetus could happen when mother could be reactivation, reinfection, it was assimptomatic and it ungiven sequele if it has been compared primer infection. That was caused maternal Ig G anti-CMVcould throug placenta and protective. Clinical symptom had been known from inflammation reaction. Oral manifestation could be vesicle and ulcer. Conclusion. CMV infection in pregnancy wasdanger infection for fetus and mother. It caused status immunity in pregnancy have been kept for prevent so that havenât infection.]]>
<![CDATA[MANIFESTASI ORAL HUMAN IMMUNE DEFICIENCY VIRUS (HIV)/ AQUIRED IMMUNO DEFICIENCY SYNDROME PADA ANAK ]]>
<![CDATA[Dewanti, I Dewa Ayu Ratna]]>
<![CDATA[ STOMATOGNATIC- Jurnal Kedokteran Gigi Vol 7, No 2 (2010) ]]>
Publisher : <![CDATA[Fakultas Kedokteran Gigi Universitas Jember ]]>
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<![CDATA[Introduction. These days HIV/AIDS infection is fast to children in the world. In developing countries such as Indonesia, this was caused first death and fourth in the world. Like at adult, HIV/AIDS to child can be manifestation in oral. Purposed of writing. This writing purposed for given information to medical and public for oral manifestation from AIDS to children. Writing benefit. This information expected to dentis can do protection and dental managemant with right. Discused. Oral manifestation HIV to children have different to adult. It may be relates to immune system in rough. Candidosis and necrotizing ulcerative gingivitis were oral lesion to child were HIV with hard immunosupresion. Linear erythema leukoplakia and sarcoma kaposi could indicator midle immunosupresion and hard. Magnification of paratis, herpes simplex virus infection and recurrent aphtous stomatitis not special relates to level ofimmunosupresion. Development of dental caries could be correlates with level of immunosupresion. Conclusion. BestManagement was by considering immunology status via precaution and inspection tooth and oral routinely formaintains health and reachs quality of good life.]]>
<![CDATA[SEPUTAR REAKSI HIPERSENSITIVITAS (ALERGI) ]]>
<![CDATA[Hikmah, Nuzulul]]>;
<![CDATA[Dewanti, I Dewa Ayu Ratna]]>
<![CDATA[ STOMATOGNATIC- Jurnal Kedokteran Gigi Vol 7, No 2 (2010) ]]>
Publisher : <![CDATA[Fakultas Kedokteran Gigi Universitas Jember ]]>
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<![CDATA[Introduction. Till now allergy was a piquancy to be submitted, because most all people have everexperienced it. Symptom and also allergen all kinds and varies can come from neither outside body nor in bodyxself. Purpose of Writing. Co-signature deeper in around allergic reaction. Discussion. Allergic reaction is immunesystem trouble marked by abundant reaction from immune response. Allergic reaction entangles allergen, is causingcells discharges matters or mediator chemistry which can destroy or hurts network in vinicity. Allergen can in the formof mote, crop powder, drug or food, is acting as antigen stimulating the happening of immune responds. Allergicreaction divided to become allergic reaction type I entangling Ig E, type II Antibody-Mediated Cytotoxi ity ( Ig G),type III ( Immune Complex Disorders) and type IV (delayed hypersensitivities). Conclusion. Allergic reaction is caused[by] allergen having manifestation to vary and divided to become reaction quickly ( type I), type II, type III and typeIV.]]>
<![CDATA[TLR4 macrophage expression of the wistar rats were inoculated by Candida albicans ]]>
<![CDATA[Dewanti, I Dewa Ayu Ratna]]>
<![CDATA[ STOMATOGNATIC- Jurnal Kedokteran Gigi Vol 7, No 3 (2010) ]]>
Publisher : <![CDATA[Fakultas Kedokteran Gigi Universitas Jember ]]>
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<![CDATA[Multidimentional crisis in Indonesia caused to multi effects. In heatlh aspect caused increase infection diseases, otherwhise mouth become some gate for entrance pathogen flora into human body. One of the most infection diseases was found in the mouth is candidiasis caused by C albicans. TLR4 were play a pivotal role in recognition of Candida albicans.The purposed experimental : This experimental purposed to know comparation of TLR4 expression macrophage of the wistar rats were inoculated C albicans. Method : The subjects of this research are 20 male-wistar rats divided into five groups : Control Group (KO) were 10 rats that were not given treatment. Experimental Groups (KP), 10 rats that were inoculated by C albicans. After 21 days, will been kill and cut on tongue for analized by imunohistochemic. The data obtained will be analyzed using t test. The result : The result this research showed that there was not a significant different (p<0,05) to comparation of TLR4 macrophage expression of Experimental Groups 1 (KP). A significant different was found at decreasing of TLR4 between KO with KP Discused : These decreasing are assumed that C albicans infection could inhibit TLR4 activities, so the decreasing of macrophage activity caused TLR4 decreased. All of those activity causes the decreasing immune response and killed activity to C albicans. The conclusion : The fact of this research is dereasing TLR4 macrophage of wistar rat was inoculated by C albicans.]]>
<![CDATA[PERAN TOLL LIKE RECEPTORS (TLRs) PADA INNATE IMMUNITY (The Role of Toll Like Receptors to Innate immunity) ]]>
<![CDATA[Hikmah, Nuzulul]]>;
<![CDATA[Dewanti, I Dewa Ayu Ratna]]>
<![CDATA[ STOMATOGNATIC- Jurnal Kedokteran Gigi Vol 8, No 1 (2011) ]]>
Publisher : <![CDATA[Fakultas Kedokteran Gigi Universitas Jember ]]>
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<![CDATA[TRLS is a homologous protein located at APC membrane cell worl as a functional receptor to activate leucocyte lead to onduce mentriger responsimun innate or inflamatory respons against palogenic againt. Functional receptor is associated wuth kinase respons to produce sitokyn and microbidal subtance. The aim of the study is to examine the respons to produce sitokyn and microbidal subtance. The aim of the study is to examine the role TLRs on innate immunity it is concluded that the role of TLRS is important functional receptor which activate leucocyte to induce mentriger respons imun innate or an inflamators respons against patogenic agent.]]>
<![CDATA[INFEKSI Cytomegalo virus (CMV) PADA IBU HAMIL DAN MANIFESTASINYA DI RONGGA MULUT ]]>
<![CDATA[I Dewa Ayu Ratna Dewanti]]>
<![CDATA[ STOMATOGNATIC - Jurnal Kedokteran Gigi Vol 7 No 1 (2010) ]]>
Publisher : <![CDATA[Fakultas Kedokteran Gigi Universitas Jember ]]>
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<![CDATA[Immune status in pregnancy was very impotant, because the effect for life to fetus andmother. If immune in pregnancy has’nt been attended, it will attack infection. Infection diseases was found in pregnancy was Cytomegalo virus (CMV) infection. Aims. This literature reviews purposes to information of CMV infection in pregnancy and manifestation in oral mucosa. Discussion. Infection in fetus could happen when mother could be reactivation, reinfection, it was assimptomatic and it ungiven sequele if it has been compared primer infection. That was caused maternal Ig G anti-CMVcould throug placenta and protective. Clinical symptom had been known from inflammation reaction. Oral manifestation could be vesicle and ulcer. Conclusion. CMV infection in pregnancy wasdanger infection for fetus and mother. It caused status immunity in pregnancy have been kept for prevent so that haven’t infection.]]>
<![CDATA[TLR4 macrophage expression of the wistar rats were inoculated by Candida albicans ]]>
<![CDATA[I Dewa Ayu Ratna Dewanti]]>
<![CDATA[ STOMATOGNATIC - Jurnal Kedokteran Gigi Vol 7 No 3 (2010) ]]>
Publisher : <![CDATA[Fakultas Kedokteran Gigi Universitas Jember ]]>
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<![CDATA[Multidimentional crisis in Indonesia caused to multi effects. In heatlh aspect caused increase infection diseases, otherwhise mouth become some gate for entrance pathogen flora into human body. One of the most infection diseases was found in the mouth is candidiasis caused by C albicans. TLR4 were play a pivotal role in recognition of Candida albicans.The purposed experimental : This experimental purposed to know comparation of TLR4 expression macrophage of the wistar rats were inoculated C albicans. Method : The subjects of this research are 20 male-wistar rats divided into five groups : Control Group (KO) were 10 rats that were not given treatment. Experimental Groups (KP), 10 rats that were inoculated by C albicans. After 21 days, will been kill and cut on tongue for analized by imunohistochemic. The data obtained will be analyzed using t test. The result : The result this research showed that there was not a significant different (p<0,05) to comparation of TLR4 macrophage expression of Experimental Groups 1 (KP). A significant different was found at decreasing of TLR4 between KO with KP Discused : These decreasing are assumed that C albicans infection could inhibit TLR4 activities, so the decreasing of macrophage activity caused TLR4 decreased. All of those activity causes the decreasing immune response and killed activity to C albicans. The conclusion : The fact of this research is dereasing TLR4 macrophage of wistar rat was inoculated by C albicans.]]>
<![CDATA[MANIFESTASI ORAL HUMAN IMMUNE DEFICIENCY VIRUS (HIV)/ AQUIRED IMMUNO DEFICIENCY SYNDROME PADA ANAK ]]>
<![CDATA[I Dewa Ayu Ratna Dewanti]]>
<![CDATA[ STOMATOGNATIC - Jurnal Kedokteran Gigi Vol 7 No 2 (2010) ]]>
Publisher : <![CDATA[Fakultas Kedokteran Gigi Universitas Jember ]]>
Show Abstract
|
Download Original
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Original Source
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<![CDATA[Introduction. These days HIV/AIDS infection is fast to children in the world. In developing countries such as Indonesia, this was caused first death and fourth in the world. Like at adult, HIV/AIDS to child can be manifestation in oral. Purposed of writing. This writing purposed for given information to medical and public for oral manifestation from AIDS to children. Writing benefit. This information expected to dentis can do protection and dental managemant with right. Discused. Oral manifestation HIV to children have different to adult. It may be relates to immune system in rough. Candidosis and necrotizing ulcerative gingivitis were oral lesion to child were HIV with hard immunosupresion. Linear erythema leukoplakia and sarcoma kaposi could indicator midle immunosupresion and hard. Magnification of paratis, herpes simplex virus infection and recurrent aphtous stomatitis not special relates to level ofimmunosupresion. Development of dental caries could be correlates with level of immunosupresion. Conclusion. BestManagement was by considering immunology status via precaution and inspection tooth and oral routinely formaintains health and reachs quality of good life.]]>
<![CDATA[PERAN TOLL LIKE RECEPTORS (TLRs) PADA INNATE IMMUNITY (The Role of Toll Like Receptors to Innate immunity) ]]>
<![CDATA[Nuzulul Hikmah]]>;
<![CDATA[I Dewa Ayu Ratna Dewanti]]>
<![CDATA[ STOMATOGNATIC - Jurnal Kedokteran Gigi Vol 8 No 1 (2011) ]]>
Publisher : <![CDATA[Fakultas Kedokteran Gigi Universitas Jember ]]>
Show Abstract
|
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<![CDATA[TRLS is a homologous protein located at APC membrane cell worl as a functional receptor to activate leucocyte lead to onduce mentriger responsimun innate or inflamatory respons against palogenic againt. Functional receptor is associated wuth kinase respons to produce sitokyn and microbidal subtance. The aim of the study is to examine the respons to produce sitokyn and microbidal subtance. The aim of the study is to examine the role TLRs on innate immunity it is concluded that the role of TLRS is important functional receptor which activate leucocyte to induce mentriger respons imun innate or an inflamators respons against patogenic agent.]]>
